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Gln is metabolized by GLS to produce alpha-ketoglutaric acid (α-KG), which serves as a crucial cofactor for TET enzymes that catalyze DNA demethylation, as well as for histone demethylases like JHDM

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notably, supplementation with PUFA-PL peroxides, but not free PUFA peroxides, restores ferroptosis in ACSL4-deficient cells, demonstrating the critical role of membrane-localized lipid peroxides in ferroptosis [34,46,47]